Abramovitch, R.B. Martin, G.B. text xx 9999 monographic und

electronic

Plant immune systems effectively prevent infections caused by the majority of microbial pathogens that are encountered by plants. However, successful pathogens have evolved specialized strategies to suppress plant defense responses and induce disease susceptibility in otherwise resistant hosts. Recent advances reveal that phytopathogenic bacteria use type III effector proteins, toxins, and other factors to inhibit host defenses. Host processes that are targeted by bacteria include programmed cell death, cell wall-based defense, hormone signaling, the expression of defense genes, and other basal defenses. The discovery of plant defenses that are vulnerable to pathogen attack has provided new insights into mechanisms that are essential for both bacterial pathogenesis and plant disease resistance. AVR, AVIRULENCE; BGH, BLUMERIA GRAMINIS F. SP. HORDEI; CDS, CELL DEATH SUPPRESSOR; COI1, CORONATINE INSENSITIVE1; COR, CORONATINE; DC3000, PSEUDOMONAS SYRINGAE PV. TOMATO STRAIN DC3000; FLS2, FLAGELLIN INSENSITIVE2; HR, HYPERSENSITIVE RESPONSE; HST, HOST-SELECTIVE TOXIN JA JASMONIC ACID JAI1 JASMONIC ACID INSENSITIVE1 NHO1 NONHOST RESISTANCE1 PCD PROGRAMMED CELL DEATH PPH PSEUDOMONAS SYRINGAE PV. PHASEOLICOLA PR PATHOGENESIS-RELATED GENE RESISTANCE SALICYLIC ACID SALICYLIC ACID XANTHOMONAS CAMPESTRIS PV. VESICATORIA https://drive.google.com/file/d/1ClDmmMQh-Eqgdv2eCmkCg3WdyqmT1NqB/view?usp=drivesdk https://drive.google.com/file/d/1ClDmmMQh-Eqgdv2eCmkCg3WdyqmT1NqB/view?usp=drivesdk 250602 20260521090959.0